Cannabis · Cigarettes · Vaping · Sexual Function

The Puff Discussion

What smoking, vaping, and cannabis do to your sexual function — and why nobody talks about it.

Nobody warns you. The health conversations around smoking and vaping focus on lungs and hearts. Cannabis conversations focus on mental health, motivation, the munchies. Almost nobody talks about what these substances do to sex.

The research exists. Here it is — plainly.

What I Notice

Something I notice — in practice and in everyday life. Cars sitting in traffic with smoke so thick inside you can barely see the driver. Couples in my consulting room, one person desperate about how much the other uses, the other defending a habit.

The habit rarely exists in isolation. There is a ritual around it — leaving the room, a specific time of day, certain people, a particular emotional state that precedes it. The rituals can be as problematic as the puffing itself.

I also notice the ethical conundrum this creates. Every person has the right to do with their body what they choose. That is not a small principle. And yet — something feels sad about a relationship lost, or never fully begun, over something that was always meant to be optional.

It is also worth noticing how much this topic gets defended. The strength of the defence is worth sitting with. Personal autonomy is a real and valid reason to protect a choice. But when the defence is particularly vigorous — when the conversation itself feels threatening — it may be worth asking what exactly is being protected.

Both sides of this conversation are fighting for something real — care, comfort, connection, autonomy, control. Understanding what each person is reaching for makes the conversation possible. What follows is condensed research on how these substances impact sexual function — not to make the decision for anyone, but to ensure that whatever decision is made, it is an informed one.

What You Inhale Goes Everywhere

The body does not compartmentalise what you put into it. Everything you inhale — every substance, every chemical, every particle — enters the bloodstream and travels to every organ, every cell, every system. There is no local effect. What reaches the lungs reaches the heart, the brain, the genitals. This is not a theory. It is how circulation works. If the circulatory system stops, everything stops. It is that fundamental.

Sexual function specifically depends on three things working together: adequate blood flow to the genitals, intact nerve signalling, and a functioning hormonal environment. The key molecule in genital blood flow is nitric oxide — it signals the smooth muscle in penile and clitoral tissue to relax, allowing blood in. Without sufficient nitric oxide, arousal becomes physiologically more difficult regardless of desire. Anything that depletes nitric oxide, disrupts nerve signalling, or alters hormonal balance will show up, eventually, in sexual experience.

Sexual function is also one of the body’s most sensitive early indicators of vascular health. Problems with arousal, erection, lubrication, and orgasm often appear before the same underlying damage becomes visible anywhere else. Most people attribute these changes to stress, age, or relationship dynamics. Sometimes the cause is simpler and more physical. If you smoke, vape, or use cannabis regularly and you have noticed changes in your sexual experience — what you are inhaling is a relevant variable. That is not a complicated idea. It is just biology.

The body does not separate what you inhale from how you function in bed. They are connected — and the connection is well documented.

Lynett Olivier
What Each One Does
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Cannabis
Weed — The Complicated One
At low to moderate doses, cannabis frequently enhances sexual experience. Increased sensory perception, reduced anxiety, heightened arousal, and more intense or prolonged orgasm are among the most consistently reported effects — particularly in women. The endocannabinoid system plays a direct role in sexual response, and THC interacts with cannabinoid receptors in areas of the brain that govern desire and arousal (Lynn et al., 2019; Kasman et al., 2020).
Lynn et al., 2019 · Sexual Medicine · Kasman et al., 2020 · Journal of Sexual Medicine
The picture for men is less consistent. Some studies report enhanced arousal and delayed ejaculation at lower doses. Others document erectile dysfunction, premature ejaculation, and inhibited ejaculation — particularly with heavier use. The research on men is genuinely conflicting and dose appears to be a significant variable (Shamloul & Bella, 2011).
Shamloul & Bella, 2011 · Journal of Sexual Medicine
Dose is the critical factor. Low to moderate doses are associated with sexual enhancement across most studies. Higher doses — and chronic heavy use — shift the picture significantly: decreased desire, difficulty reaching orgasm, reduced motivation for partnered sex, and in men, increased risk of ejaculatory problems. The same substance that enhances at one level impairs at another (PMC, 2024).
PMC Review, 2024 · Psychopharmacology
Around one in three users report that cannabis makes them withdraw into themselves during sex — losing erotic connection to their partner, becoming too internal to remain present. If that is your experience, the research is clear: cannabis is not helping your sexual life, regardless of what it feels like in the moment.
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Cigarettes
Tobacco — The Slow Damage
Smoking is an independent risk factor for erectile dysfunction — one of the most documented modifiable causes. Smokers are approximately twice as likely to experience moderate to severe erectile dysfunction as non-smokers, independent of age and other health conditions. The mechanism is vascular: cigarette smoke directly inhibits nitric oxide synthase, depleting the molecule the body uses to enable erection (Cao et al., 2013; Allen & Tostes, 2023).
Cao et al., 2013 · meta-analysis · Allen & Tostes, 2023 · Sexual Medicine Reviews
The damage is dose-dependent and cumulative. Men who smoke more than 20 cigarettes per day have double the risk of severe erectile dysfunction compared to lighter smokers. The longer the smoking history, the greater the risk. This is not acute — it builds over years.
McVary et al., 2001 · Journal of Urology
Nicotine alone impairs erectile response — acutely, even in non-smokers. A controlled trial demonstrated that a single intermediate dose of isolated nicotine significantly reduced erectile response in healthy young men who had never smoked. The mechanism is direct: nicotine disrupts the autonomic nervous system signalling that governs genital blood flow (Harte & Meston, 2008).
Harte & Meston, 2008 · PMC
Smoking is also linked to reduced libido in men — with current smokers showing higher rates of low sex drive compared to non-smokers and former smokers. Nicotine addiction intensifies anxiety and depression, both of which suppress desire. The sexual cost of smoking is not limited to mechanics — it affects motivation too (Sooriyamoorthy & Leslie, 2022).
Sooriyamoorthy & Leslie, 2022 · BJU International
Smoking reduces genital sensitivity and orgasm quality — often without the person noticing. Because the effect accumulates gradually, many people attribute reduced arousal, weaker orgasms, or lower orgasm frequency to age, stress, or relationship factors. Controlled research shows nicotine produces a measurable reduction in physiological genital arousal — up to 30% attenuation in women in one study — and that lower clitoral blood flow correlates directly with reduced orgasm frequency. The person rarely notices a sudden change. They simply enjoy sex a little less than they once did, without knowing why (Harte & Meston, 2008; Battaglia et al., 2008).
Harte & Meston, 2008 · Journal of Sexual Medicine · Battaglia et al., 2008 · Journal of Sexual Medicine
There is meaningful evidence that quitting improves erectile function — particularly in younger men without significant vascular damage already in place. Former smokers show markedly better erectile function and libido than current smokers. The body, where it can, recovers.
Allen & Tostes, 2023 · Sexual Medicine Reviews
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Vaping / E-cigarettes
Vaping — The False Safety
Vaping and e-cigarettes are the same thing — e-cigarette is the clinical and regulatory term, vaping is what people actually call it. The device heats a liquid containing nicotine and produces an aerosol.
Vaping delivers the same primary mechanism of harm as cigarettes — nicotine. The vasoconstriction that nicotine causes, and the disruption to nitric oxide availability, occurs whether the delivery system is a cigarette or a vape. The assumption that switching from cigarettes to vaping eliminates sexual health risk is not supported by the evidence.
Allen & Tostes, 2023 · Sexual Medicine Reviews
Lower concentration per puff does not mean lower overall exposure. Many pod-based vapes deliver the nicotine equivalent of 20 cigarettes in a single pod. A cigarette burns down and ends — a vape does not. Because there is no natural endpoint, vaping sessions are longer, more frequent, and often continuous throughout the day. Vaping is also used indoors, in cars, at desks, in contexts where smoking was never permitted — meaning it happens more. The net nicotine exposure for a heavy vaper may equal or exceed that of a cigarette smoker, despite each puff being “less.” The frequency is the point.
Harte & Meston, 2008 · PMC · Allen & Tostes, 2023 · Sexual Medicine Reviews
Cigarettes cause additional damage beyond nicotine — tar and combustion products harm the endothelium in ways vaping does not replicate to the same degree. This is why cigarettes are genuinely more harmful overall. But this is not an argument for vaping. It is an argument against cigarettes that does not make vaping safe.
Pizzol et al., 2020 · PubMed
The sensitivity and orgasm effect applies to vaping too. Since the mechanism is nicotine-driven vasoconstriction — the same molecule, the same pathway — reduced genital sensitivity, decreased lubrication, and lower orgasm frequency are documented in vapers as in smokers. Daily vapers are 2.4 times more likely to experience erectile dysfunction than those who have never vaped (El-Shahawy et al., 2022). The delivery device is different. The sexual health cost is not.
El-Shahawy et al., 2022 · American Journal of Preventive Medicine
Vaping among young people is particularly relevant. The perception of safety has driven uptake in an age group that would not have smoked cigarettes — meaning the long-term vascular effects are now accumulating in a population that believes it is not at risk. The sexual health consequences of this may not become visible for years. The long-term data on vaping specifically is still limited — which is not reassurance. It is a gap.
What Stopping Does

For cigarettes and vaping, the evidence on cessation is meaningful. Former smokers show significantly better erectile function and libido than current smokers. The endothelium — the lining of blood vessels responsible for nitric oxide production — begins to recover after smoking cessation, and erectile function improves in those who quit, particularly men who are younger and whose vascular damage has not yet become structural (Allen & Tostes, 2023). This is not guaranteed, and it is not immediate. But it is documented.

For cannabis, the picture depends entirely on how you are using it. If low to moderate use is enhancing your sexual experience without driving dependency or withdrawal from your partner, the research does not suggest you need to stop. If heavy or chronic use has shifted the picture — reduced desire, difficulty orgasming, increased withdrawal into yourself during sex — reducing use is the clinically supported direction.

Worth knowing

The vascular system has a remarkable capacity to recover when the damage stops accumulating. Sexual function often improves in ways people did not expect — over months — after quitting smoking.

The body is not vindictive. It responds to what you give it.

Why Nobody Talks About This

Sexual function has not historically been considered a key element of human functioning, connection, or fulfilment — not in medicine, not in public health, and not in most cultural conversations about wellbeing. It has been treated as a private matter, and a secondary one. How you enjoy sex, whether you enjoy it, desire, lust, disgust — these have not been considered important enough to base decisions on. Even now, in an era that speaks more openly about mental health and wellbeing than any before it, the desire for a fulfilling sexual life can still feel like the lesser need. For some, it is considered a bonus if anything — something that will in any event wane over time. They never consider that other things they give precedence to in the present will also wane over time.

There is also the question of medical priority. A practitioner treating someone for smoking-related cardiovascular disease, lung damage, or cancer is not indifferent to that person’s sexual function. They likely know it is being affected. But their first priority — and in many cases their only priority — is not the quality of your life. It is that you have one. Heart disease kills. Sexual dysfunction does not. So the practitioner addresses what is life-threatening, and the quality-of-life conversation gets deprioritised. This is not negligence. It is triage.

What that means in practice is that the onus falls on the individual. And many people never pick it up — not because they don’t care about their sexual health, but because nobody has told them there is anything to pick up. The connection between what they inhale and how they function in bed has simply never been part of the conversation they were offered.

What Now?

We live in an era of extraordinary health consciousness. People track their sleep, scrutinise ingredients, and consider carefully what goes into their bodies. And then vape between meetings, smoke without a second thought, or reach for a joint to wind down — without connecting it to how the body functions. There are no free choices where the body is concerned. Every choice has a cellular consequence.

If this article has landed somewhere, the most useful thing you can do is start with two questions rather than a plan.

What created it? Not the habit itself, but what was underneath it when it started. Some habits form because difficult emotions needed somewhere to go. Some form through social context — a desire to belong, to fit in, to be part of something. Some form through genetic predisposition — an experiment that became a dependency before the person fully understood what was happening. The origin matters because it shapes everything that follows.

What keeps it in place now? What the substance is currently doing for you — what it regulates, what it eases, what gap it fills. A habit that began as social belonging may now be managing anxiety. A habit that began recreationally may now be the only way to sleep. And sometimes, even when the original driver is long gone, the neurological pathway remains — the habit outlives the reason it started. The brain has been conditioned. Understanding what maintains it is more useful than trying to stop it without that understanding.

It is also worth naming something the research consistently identifies: most people who use substances problematically are not unaware. They are in two minds. That ambivalence — wanting to stop and not wanting to, knowing the cost and continuing anyway — is not weakness or contradiction. It is the most common presentation there is. Naming it honestly is usually the first step through it.

The work follows from both of those answers. That work looks different for every person — and that is precisely why a conversation with someone equipped to help is worth more than a plan you found in an article.

A Closing Word

The body is not a collection of separate systems. It is one organism, running on one circulatory system, responding to everything you give it — or withhold from it. What you inhale reaches every cell. What reaches every cell shapes how every system functions. Including the one that governs your capacity for pleasure, connection, and intimacy.

You deserve a body that functions fully. That is not a small thing to consider.

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References
Allen, M. S., & Tostes, R. C. (2023). Cigarette smoking and erectile dysfunction: An updated review with a focus on pathophysiology, e-cigarettes, and smoking cessation. Sexual Medicine Reviews, 11(1), 61–73.  |  Cao, S., et al. (2013). Cigarette smoking and risk of erectile dysfunction: Systematic review of observational studies with meta-analysis. Journal of Sexual Medicine.  |  Harte, C. B., & Meston, C. M. (2008). Acute effects of nicotine on physiological and subjective sexual arousal in nonsmoking men. PMC.  |  McVary, K. T., et al. (2001). Smoking and erectile dysfunction: Evidence based analysis. Journal of Urology, 166(5), 1624–1632.  |  Pizzol, D., et al. (2020). People smoke for nicotine, but lose sexual and reproductive health for tar. PubMed.  |  Sooriyamoorthy, T., & Leslie, S. W. (2022). The impact of smoking on sexual function. BJU International.  |  Lynn, B. K., et al. (2019). The relationship between marijuana use prior to sex and sexual function in women. Sexual Medicine, 7(2), 192–197.  |  Kasman, A. M., et al. (2020). Association between cannabis use and sexual frequency in the United States. Journal of Sexual Medicine, 17(6).  |  Shamloul, R., & Bella, A. J. (2011). Impact of cannabis use on male sexual health. Journal of Sexual Medicine, 8(4).  |  PMC Review. (2024). Update on cannabis in human sexuality. Psychopharmacology.  |  Lynn, B., et al. (2020). Effects of cannabinoids on female sexual function. Sexual Medicine Reviews, 8(1), 18–27.  |  El-Shahawy, O., et al. (2022). Association of e-cigarettes with erectile dysfunction: The Population Assessment of Tobacco and Health Study. American Journal of Preventive Medicine, 62(1), 26–38.  |  Battaglia, C., et al. (2008). Clitoral vascularization and sexual behavior in young patients treated with drospirenone-estradiol or contraceptive vaginal ring. Journal of Sexual Medicine, 5(6).